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I hope the few sources below answer your question. I strongly recommend you read actual sources as opposed to the parts I have quoted. I don't know much about this topic so I did not attempt to translate anything in laymen terms, since I might have distorted or misinterpreted information in the process.
However, the three sources below are one of the best-known studies on the genetic basis of ADHD. I have quoted the two sources in direct response to your question about the proposed role of dopamine and serotonin transporter genes. Percentage due to variation attributed to heritability Twin, family and adoption studies have suggested that there is a strong genetic contribution to ADHD, with a mean heritability estimate of 76% However, it has been difficult to implicate any specific gene in ADHD beyond reasonable doubt. The difficulties encountered in identifying genetic variants increasing the risk for ADHD could be at least in part owing to the heterogeneity and complexity of this clinical syndrome.Consequently, it has been proposed that the use of intermediate phenotypes (or endophenotypes) relevant to ADHD are likely to be more informative than the DSM-IV1 diagnoses, allowing for increased detection of genetic effects. Genome-wide linkage scans To date, seven genome-wide ADHD linkage scans have been published and some chromosome regions such as 5p13, 14q12, and 17p11 have been indicated in multiple studies [Fisher et al., 2002; Bakker et al., 2003; Arcos-Burgos et al., 2004; Hebebrand et al., 2006; Ogdie et al., 2006; Faraone et al., 2007; Asherson et al., 2008; Romanos et al., 2008].
However, no chromosome region has been consistently identified across the scans and the majority of the findings were unique to each study. This is not unexpected because the power of individual scans is likely to be low for a complex trait such as ADHD which may only have genes of small to moderate effects [Risch and Merikangas, 1996; Waldman and Gizer, 2006]. Dopamine transporter (DAT1) The DA transporter (DAT1) gene harbours a 40-bp repeat sequence variable number tandem repeat polymorphism located in the 3′-untranslated region. The most common alleles are the 10- (480-bp, 71.9%) and 9-repeat (440-bp, 23.4%) alleles, although these frequencies are variable from one population to the next. Homozygosity for the 10-repeat allele was reported to be associated with higher DA transporter protein in the striatum, a region where it is heavily expressed56 and where it serves as the primary means of DA reuptake.